Vaccine injury testimony – Another incredible revelation coming from Marcella Piper-Terry of VaxTruth.Org

Vaccine injury testimony – Another incredible revelation coming from Marcella Piper-Terry of VaxTruth.Org. An employee of a medical billing company is sending records showing fetal death occurring following flu and TDaP vaccines when administered to pregnant women.

Polio provocation: solving a mystery with the help of history

The art of medicine
Polio provocation: solving a mystery with the help of history
In 1998, State University of New York researchers Matthias Gromeier and Eckard Wimmer published a pioneering article on the mechanism of injection-induced polio paralysis. Through their laboratory work, they discovered that tissue injury produced by an injection aided the poliovirus to infect the body and readily journey to the spinal cord. For the first time, health professionals working in polio endemic regions had scientific evidence that paediatric injections could incite paralysis.
Substantiation of the theory seemed to vindicate the cautious policies of the 1950s and the importance of maintaining herd immunity against polio. In countries where the virus was controlled through vaccination programmes, the risk of polio provocation was insignificant. However, in regions where polio was endemic, immunisation sequence mattered until eradication of the virus was achieved. The discovery showed that polio vaccination was vital to the wider success of public health programmes and that it needed to be undertaken before other paediatric immunisations to reduce the risk of provoking polio.
One of the first medical procedures implicated in the causation of polio was tonsil surgery. A study of more than 2000 case histories in the 1940s by the Harvard Infantile Paralysis Commission concluded that tonsillectomies led to a significant risk of respiratory paralysis due to bulbar polio. Although proponents of the theory did not entirely oppose tonsillectomies, they cautioned that such interventions should be avoided during epidemics. Reflecting the growing body of evidence that tonsillectomies could provoke polio, many doctors in the USA adjusted their surgical procedures to account for disease-endemic factors. “The policy of the United States Army”, Major-General E A Noyes acknowledged in 1948, “has been to stop tonsil and adenoid operations during epidemics”. Even though laboratory technology at the time was not sufficiently advanced to unravel the mechanism, published evidence affected clinical practice.
Concerns about tonsillectomies coincided with indications that paediatric injections could also incite polio paralysis. Evidence of this correlation was first published by German doctors, who noted that children who had received treatment for congenital syphilis later became paralysed in the injected limb. Although further studies from Italy and France corroborated this link, it was not until the end of World War II that injection-induced polio emerged as a public health concern.
The application of epidemiological surveillance and statistical methods enabled researchers to trace the steady rise in polio incidence along with the expansion of immunisation programmes for diphtheria, pertussis, and tetanus. A report that emerged from Guy’s and Evelina Hospitals, London, in 1950, found that 17 cases of polio paralysis developed in the limb injected with pertussis or tetanus inoculations. Results published by Australian doctor Bertram McCloskey also showed a strong association between injections and polio paralysis. Meanwhile, in the USA, public health researchers in New York and Pennsylvania reached similar conclusions. Clinical evidence, derived from across three continents, had established a theory that required attention.
Several ideas were posited by health professionals in an effort to understand how immunisations for diphtheria, tetanus, and pertussis seemed to provoke polio infection. One theorist posited that injections injured human tissues and predisposed them to viral infection. A further theory advanced by Harold K Faber of the Stanford University School of Medicine argued that the ubiquitous poliovirus, already present on the skin of many children, was being driven into the body during immunisations and thus seeded deep into the tissue.
Meanwhile, American newspapers advised parents to postpone vaccinations during warm weather or epidemics, citing evidence that some children developed polio within a month of injection. As debates swirled and publicity mounted, parents were asked to weigh the potential risks of immunisations with their benefits.
… the American Public Health Association, accommodated the possibility of polio provocation and encouraged health professionals to avoid “indiscriminate” injections and “booster shots” during epidemics.
Most health professionals reformed their immunisation practices and accepted that seasonal factors and cycles of disease were important to consider before immunising children.
Although medical scientists failed to understand the epidemiological mechanism behind polio provocation, the Salk and Sabin vaccines pushed the issue to the margins of clinical attention.

Chronic Neuroimmune Diseases
Chronic Fatigue Syndrome
A polio by another name
But a body of evidence is growing linking Chronic Fatigue Syndrome (CFS), also called myalgic encephalomyelitis (ME), to this terrible disease, largely caused by attempts to eradicate polio. An alternative polio seems to be upon us.
The proceedings of the first intemational scientific conference on the Post-Polio Syndrome in the US have been collated in the Annals of the New York Academy of Science. It includes 50 papers written by 118 contributors from a wide range of specialties, including clinical neurology.
In particular, papers by Dr Richard Bruno, assistant professor at the New Jersey Medical School’s department of physical medicine and rehabilitation and director of Post-Polio Rehabilitation and Research Service at the Kessler Institute for Rehabilitation in New Jersey, and four other specialists compare Chronic Fatigue Syndrome and Post-Polio Syndrome (Dalakas, et al, ed. The Post Polio Syndrome: Advances in the Pathogenesis Treatment,Annals, NY Academy, Sciences, 1995: 273: 1-409). Post-Polio is developing in those who had polio 25-30 years previously. Clinically, it is indistinguishable from CFS.
Other researchers demonstrate that CFS is just another form of polio, which has increased with the advent of polio vaccination. As one type of gut virus has been eradicated, so other forms have had the space to proliferate. Up to one in every 500 Americans may have CFS, according to the Centers for Disease Control.
To understand the link one needs to understand the microbiological habits of both polio and other enterovirus disease-that is, gut bugs.
A historical accident has led to various names being given to viruses, all of which share physical , chemical and epidemiological characteristics of what we consider the classic polio virus, which science refers to as polio viruses 1, 2, and 3 (Dowsett: Journal of Hospital Infection, 1988:11:103-15). ln 1948, a polio-like illness in New York state prompted scientists to culture the virus. But what grew looked to them at that time like a new virus.
They called it “Coxsackie’ after the small town up the Hudson River where it was found. And they called the disease “Atypical Polio” because its symptoms identified it as a kind of polio, despite the virus being apparently different.
This kind of polio, “Atypical Polio,’ has since been renamed, ‘Chronic Fatigue Syndrome,’ or ME. But it remains a kind of poIio despite the change of name. and newer technology has shown up the generic similarities of the most frequent agent that causes it.
These techniques place Coxsackie, the virus most often implicated in CFS, in the polio family tree, along with so-called echo viruses. Coxsackie has been further divided into Coxsackie type A (with 24 viruses) and Coxsackie type B (six viruses ). There are 34 echo viruses. In total, there are at least 72 enteroviruses in all, with new ones still being discovered.
All this has been unnecessarily confusing and complicated, even for doctors. These days newly discovered enteroviruses are just given a new number, not a new name, since their inter-relationship is recognized.
Had the techniques been available that we now have at our disposal, all these viruses might simply have been called “Polio 1 through 72.”

Dr. Mark Geier discusses aspects of the flu vaccine

Dr. Mark Geier discusses aspects of the flu vaccine

More deaths among otherwise healthy people are being reported all across the United States among children and adults who received this year’s flu vaccination. Here are a couple of the latest deaths being reported in local media stations

CDC: Current Flu Vaccine Not a Good Match for This Season’s Viruses, Ineffective

Kiera, Ayzlee, Amber, and Kristie had two things in common. They all got this year’s flu shot. They all were diagnosed with Type A Influenza, which is one of the Influenza strains contained each year in the flu shot, regardless of which version is given. Influenza Type B is also contained in yearly flu shots. Ayzlee was diagnosed with both Type A and Type B Influenza. Because of her age, it is likely that Ayzlee received the Flu Mist vaccine – a live virus vaccine. We do not know for sure which vaccines they received. All we know is that each of these formerly healthy, vibrant individuals got the flu shot. They (or their parents) thought they were protected and now they are gone.

Patrick Driscoll said that Kiera had been vaccinated against the flu. He said doctors confirmed that Kiera had contracted the same strain for which she had been vaccinated.

37 Year Old “Healthy” Woman Dies from Flu, Even Though She Received the Flu Shot

Another flu death of an otherwise healthy person after receiving the flu vaccine has been reported in Wisconsin. WISN in Wisconsin is reporting that 26-year-old Katherine McQuestion has died from flu complications, after she received the flu shot. Katherine was reportedly a newlywed, and was required to receive the flu shot as part of her employment. She was a radiology technician and worked at St. Catherine’s Medical Center in Pleasant Prairie, Wisconsin according to WISN.

Another Precious Child Falls Victim to the California Vaccine Mandates

Another Precious Child Falls Victim to the California Vaccine Mandates

MMRV Vaccine made my child disabled in 3 days.
I am a desperate parent looking for local and social media to help me to investigate my 6 year old child losing her ability to coordinate eyes just 3 days after MMRV vaccine.
In the last week, my daughter was misdiagnosed, let go untreated from hospital totaling in 4 visits and just got released with “maybe” hopes to regain her normal vision. I am puzzled and torn apart how on 4/14/16 child that has been examined by her pediatrician and found perfectly healthy end up being disabled after MMRV vaccine in 3 days.
Please help me bring awareness of this incident and locate medical professional to help my daughter to regain her vision back. Thank you!”

Study suggest that 6th nerve paralysis is a known condition and had been documented in connection to vaccination

Recurrent 6th nerve palsy in a child following different live attenuated vaccines: case report
Background
Recurrent benign 6th nerve palsy in the paediatric age group is uncommon, but has been described following viral and bacterial infections. It has also been temporally associated with immunization, but has not been previously described following two different live attenuated vaccines.

Conclusions
The majority of benign 6th nerve palsies do not have a sinister cause and have an excellent prognosis, with recovery expected in most cases. The exact pathophysiology is unknown, although hypotheses including autoimmune mechanisms and direct viral invasion could explain the pathophysiology behind immunization related nerve palsies. It is important to rule out other aetiologies with thorough history, physical examination and investigations. There is limited information in the literature regarding the safety of a repeat dose of a live vaccine in this setting. Future immunizations should be considered on a case-by-case basis.

Hepatitis B Vaccine for Infants: Is it Worth It?
Here are some studies that have investigated the health effects of hepatitis B vaccine in U.S. children:

Controlling for age, race and gender simultaneously in the 1994 NHIS, hepatitis B vaccine was found to be associated with prevalent arthritis [odds ratio (OR) = 5.91], incident acute ear infections (OR = 1.60), and incident pharyngitis/nasopharyngitis (OR = 1.41).

Hepatitis B vaccinated children had an unadjusted odds ratio of 2.94 and age-adjusted odds ratio of 2.35 for liver problems compared with non-hepatitis B vaccinated children in the 1993 National Health Interview Survey. Hepatitis B vaccinated children had an unadjusted odds ratio of 2.57 and age-adjusted odds ratio of 1.53 for liver problems compared with non-hepatitis B vaccinated children in the 1994 National Health Interview Survey Dataset.

In people not previously exposed to hepatitis B, vaccination has unclear effect on the risk of developing infection, as compared to no vaccination. The risk of lacking protective antibody levels as well as serious and non-serious adverse events appear comparable among recipients and non-recipients of hepatitis B vaccine. (no benefit from receiving the vaccine)

Engerix B vaccine appears to increase [risk of CNS inflammatory demyelination], particularly for confirmed multiple sclerosis, in the longer term.

This study investigated the association between vaccination with the Hepatitis B triple series vaccine prior to 2000 and developmental disability in children aged 1-9 years (n=1824), proxied by parental report that their child receives early intervention or special education services (EIS)… The odds of receiving EIS were approximately nine times as great for vaccinated boys as for unvaccinated boys…This study found statistically significant evidence to suggest that boys in the United States who were vaccinated with the triple series Hepatitis B vaccine, during the time period in which vaccines were manufactured with thimerosal, were more susceptible to developmental disability than were unvaccinated boys.

Boys vaccinated as neonates had threefold greater odds for autism diagnosis compared to boys never vaccinated or vaccinated after the first month of life. Findings suggest that U.S. male neonates vaccinated with the hepatitis B vaccine prior to 1999 (from vaccination record) had a threefold higher risk for parental report of autism diagnosis compared to boys not vaccinated as neonates during that same time period.

The last two articles listed above involved data analysis for children who received the hepatitis B vaccine prior to the elimination of thimerosal, or mercury from the vaccine. This may lead you to believe once that was accomplished the problem was solved. The current hepatitis B vaccines contain high amounts of aluminum. The following articles have to do with the problems associated with aluminum in infant vaccines:

In the present review we describe how the use of a systems biology approach in cultured hepatoblastoma cells (HepG2) allowed the identification of the molecular targets of Al toxicity. Mitochondrial metabolism is the main site of the toxicological action of Al. Fe-dependent and redox sensitive enzymes in the tricarboxylic acid (TCA) cycle and oxidative phosphorylation (OXPHOS) are dramatically decreased by Al exposure. In an effort to compensate for diminished mitochondrial function, Al-treated cells stabilize hypoxia inducible factor -1a (HIF-1a) to increase ATP production by glocolysis. Additionally, Al toxicity leads to an increase in intracellular lipid accumulation due to enhanced lipogenesis and a decrease in the B-oxidation of fatty acids. Central to these effects is the alteration of a-ketoglutarate (KG) homeostasis. In Al-exposed cells, KG is preferentially used to quench ROS leading to succinate accumulation and HIF-1a stabilization. Moreover, the channeling of KG to combat oxidative stress leads to a reduction of L-carnitine biosynthesis and a concomitant decrease in fatty acid oxidation. The fluidity and interaction of these metabolic modules and the implications of these findings in liver-related disorders are discussed herein.

(Note: The importance of these findings is relevant especially for brain development and for chronic autoimmune disorders such as diabetes and autism. The disruption of mitochondrial metabolism is significant in the increased risk of vaccine-injury in persons who have underlying mitochondrial disorders.)

When assessing adjuvant toxicity in children, several key points ought to be considered:

(i) infants and children should not be viewed as “small adults” with regard to toxicological risk as their unique physiology makes them much more vulnerable to toxic insults;
(ii) in adult humans Al vaccine adjuvants have been linked to a variety of serious autoimmune and inflammatory conditions (i.e., “ASIA”), yet children are regularly exposed to much higher amounts of Al from vaccines than adults;
(iii) it is often assumed that peripheral immune responses do not affect brain function. However, it is now clearly established that there is a bidirectional neuro-immune cross-talk that plays crucial roles in immunoregulation as well as brain function. In turn, perturbations of the neuro-immune axis have been demonstrated in many autoimmune diseases encompassed in “ASIA” and are thought to be driven by a hyperactive immune response;
and
(iv) the same components of the neuro-immune axis that play key roles in brain development and immune function are heavily targeted by Al adjuvants. In summary, research evidence shows that increasing concerns about current vaccination practices may indeed be warranted. Because children may be most at risk of vaccine-induced complications, a rigorous evaluation of the vaccine-related adverse health impacts in the pediatric population is urgently needed.

With all of these concerns about the safety of aluminum in vaccines, and about the Hepatitis B vaccine in particular, you may be led to ask yourself, “Is it worth it?”

Another JP Morgan Banker Leaps to His Death

Another JP Morgan Banker Leaps to His Death

Hong Kong man becomes 7th banker to die under mysterious circumstances

Paul Joseph Watson

Infowars.com February 18, 2014

Yet another banker has committed suicide, with a JP Morgan forex trader leaping to his death from the top of the firm’s Chater House headquarters in Hong Kong

Over the past few weeks at least seven bankers have died under mysterious circumstances, including another JP Morgan senior manager who jumped off the top of a skyscraper in London last month.

Speculation is rife that the series of deaths are connected to some kind of looming financial crisis or a huge legal case targeting bankers for malfeasance, although no definite link has been established.

Eyewitnesses said that the man, who was in his 30′s, accessed the roof of the 30 story office tower and jumped, with police on the scene failing to talk him out of committing suicide. Chater House is JP Morgan’s main regional Asian office.

“According to several JP Morgan employees, the man was a forex trader with the company,” reports the South China Morning Post, adding that his surname was Lee. The bank itself refused to confirm that the man was an employee.

Lee becomes the 7th banker to suddenly die in recent weeks. Questions as to whether the deaths are merely a coincidence or are linked to some as yet unknown factor continue to swirl.

– On January 26, former Deutsche Bank executive Broeksmit was found dead at his South Kensington home after police responded to reports of a man found hanging at a house. According to reports, Broeksmit had “close ties to co-chief executive Anshu Jain.”

– Gabriel Magee, a 39-year-old senior manager at JP Morgan’s European headquarters, jumped 500ft from the top of the bank’s headquarters in central London on January 27, landing on an adjacent 9 story roof.

– Mike Dueker, the chief economist at Russell Investments, fell down a 50 foot embankment in what police are describing as a suicide. He was reported missing on January 29 by friends, who said he had been “having problems at work.”

– Richard Talley, 57, founder of American Title Services in Centennial, Colorado, was also found dead earlier this month after apparentlyshooting himself with a nail gun.

– 37-year-old JP Morgan executive director Ryan Henry Crane died last week.

– Tim Dickenson, a U.K.-based communications director at Swiss Re AG, also died last month, although the circumstances surrounding his death are still unknown.