Vaccine news – CDC Knew Its Vaccine Program Was Exposing Children to Dangerous Mercury Levels Since 1999

World-famous scientist issues warning about genetically modified vaccines
Here’s a look at just some of the genetically modified vaccines on the market today — and the risks we know about:
RotaTeq: This is supposed to protect babies from rotavirsues, and lots of kids get three doses before they even reach six months of age. But it’s actually made from a cross between cow and human DNA, and clinical trials found that infants are twice as likely to suffer seizures within the first two weeks after they get the vaccine.
Flucelvax: This new flu jab is being marketed like crazy this time of year. But what they’re not telling you is that it was actually made by combining human flu strains with kidney cells from a cocker spaniel! And while Flucelvax has only been around for a couple years, we already know that injecting ourselves with dog DNA is bad news. The vaccine nearly doubles your risk of a painful muscle condition called myalgia.
HPV vaccines, like Gardasil: I’ve told you stories about healthy, young girls who ended up paralyzed, unable to feed themselves and incapable of even attending school shortly after getting these shots. And currently, Virginia, Rhode Island and the District of Columbia require a Gardasil shot just to attend school.
And there are a whole bunch of new genetically-spliced recombinant vaccines coming down the pike, including one for measles.
Every time scientists try to warn us about the dangers of vaccines, the mainstream bends over backwards to call them quacks. But, trust me, nobody is saying that about Stephen Hawking.
Sources:
“Most threats to humans come from technology, warns Hawking” Ian Sample, January 19, 2016, The Guardian, msn.com

Vaccines Licensed for Use in the United States

Stephen Hawking Says the Human Race is in Danger and it’s our Own Fault
I feel so smart today. It isn’t often that the scientific merit of my ideas is confirmed by Stephen Hawking.   Today, there is an article in The Guardian, reposted in the MSN news, titled, “Most threats to humans come from science and technology, warns Hawking”. Excerpt:
“Speaking to the Radio Times ahead of the BBC Reith Lecture, in which he will explain the science of black holes, Hawking said most of the threats humans now face come from advances in science and technology, such as nuclear weapons and genetically engineered viruses.”
Genetically engineered viruses? Where have I heard that before? Oh, that’s what drug companies put into vaccines that doctors, nurses and pharmacists inject directly into and/or put in the mouths of people – vaccines for illnesses that include Hepatitis B, HPV, flu, and rotavirus.
The vaccine for Hepatitis B was the first to be made from a genetically engineered virus as announced in this New York Times article from July 1986 (please see – the reader will appreciate the historical significance). Note who, in 1986, the Hepatitis B vaccine was recommended for:
Dr. Young recommended vaccinations with either hepatitis vaccine for dental and medical workers, susceptible homosexuals and drug users, the newborn children of infected women, and, among other groups, travelers to parts of the world where hepatitis B is rampant, such as southeast Asia, sub-Saharan Africa, and parts of the Middle East.
That was a couple of years before they got the no doubt economically motivated crazy idea to assault every baby born in this country with the full series of this genetically modified concoction, the first dose of which would be given by their decree when the baby is in medical custody, in hospital, within 12 hours of birth.

ALUMINUM contained in vaccines is a metalloestrogen
Study – Metalloestrogens: an emerging class of inorganic xenoestrogens with potential to add to the oestrogenic burden of the human breast.
J Appl Toxicol. 2006 May-Jun;26(3):191-7.
Abstract
Many compounds in the environment have been shown capable of binding to cellular oestrogen receptors and then mimicking the actions of physiological oestrogens. The widespread origin and diversity in chemical structure of these environmental oestrogens is extensive but to date such compounds have been organic and in particular phenolic or carbon ring structures of varying structural complexity. Recent reports of the ability of certain metal ions to also bind to oestrogen receptors and to give rise to oestrogen agonist responses in vitro and in vivo has resulted in the realisation that environmental oestrogens can also be inorganic and such xenoestrogens have been termed metalloestrogens. This report highlights studies which show metalloestrogens to include aluminium, antimony, arsenite, barium, cadmium, chromium (Cr(II)), cobalt, copper, lead, mercury, nickel, selenite, tin and vanadate. The potential for these metal ions to add to the burden of aberrant oestrogen signalling within the human breast is discussed.

13 Year-Old Boy Permanently Disabled from Chicken Pox Vaccine Wins His Case in Vaccine Court
A young man was recently awarded compensation in the United States Court of Federal Claims Vaccine Court, for injuries he sustained after being administered the hepatitis A and varicella vaccinations in 2009.  After five long years of litigation, Health and Human Services (HHS), the Respondent in all vaccine injury cases, conceded that the varicella vaccination did in fact cause RD’s vaccine injury, transverse myelitis, which has left him a tetraplegic.
In November 2014, HHS conceded that the vaccination caused RD’s injuries.  Even with this concession, his case continued for another year in the damages phase, during which time the parties continued to negotiate the amount of damages that RD would receive for his injuries.  Although he was compensated for his suffering and injuries, the monetary award will never compensate for the lifelong effects this young man is suffering from his vaccine injury.
Five Long Years
RD was only 13 when his life changed forever.  At a routine well-child visit in 2009, the doctor informed RD’s parents that he was due to receive the hepatitis A and varicella vaccinations.  His parents complied with the doctor’s order and RD received the vaccinations.
RD’s mother explained that, at that time in RD’s state, only one dose of varicella vaccine was required and RD had already received one dose of that vaccine.  This second dose that was administered to RD at this well visit was determined to be the cause of RD’s horrific injuries, and it was not even required for him, which his family didn’t realize until it was too late.
About 14 days later, RD began to experience excruciating pain shooting through his body along with tingling, numbness and paralysis of his limbs. After extensive testing and many invasive procedures, RD was diagnosed with transverse myelitis.
RD’s parents filed a case in Vaccine Court, which took over five years to settle. RD and his family faced arduous heartbreak along the way. In the ruling, a representative from the United States Department of Justice agreed that “a preponderance of the evidence establishes that petitioner’s transverse myelitis was caused-in-fact by the administration of his August 12, 2009 varicella vaccine.” [1]
RD’s lawyer, Patricia Finn, stated that:
“The injuries that RD suffered from this vaccine are severe and lifelong.  Even though he has received a significant award as far as the awards in the Vaccine Court go, no amount of money will ever compensate him for what he has lost.
But RD is an amazing young man who has not let this injury stop him in any way.  He has graduated high school with his class, attends a Tier 1 college, and has great aspirations that I know he will achieve despite the challenges he faces because of his injuries.”
RD’s Immune System Attacked His Spine

Ignoring the agency’s own scientific evidence, the CDC’s webpage stubbornly insists that the “two types of mercury to which people may be exposed—methylmercury and ethylmercury—are very different.” The new CDC study directly contradicts this assertion, “There are many commonalities/similarities in the mechanisms of toxic action of methylmercury and ethylmercury …”
The study meticulously details identical toxicity pathways shared by both forms of mercury:
Both ethyl and methyl mercury cause DNA damage or impair DNA synthesis (Burke et al. 2006; Sharpe et al. 2012; Wu et al. 2008).
Both cause oxidative stress/creation of reactive oxygen species (Dreiem and Seegal 2007; Garg and Chang 2006; Myhre et al. 2003; Sharpe et al. 2012; Yin et al. 2007).
Both decrease glutathione activity, thus providing less protection from the oxidative stress caused by MeHg and EtHg (Carocci et al. 2014; Ndountse and Chan (2008); Choi et al. 1996; Franco et al. 2006; Mori et al. 2007; Muller et al. 2001; Ndountse and Chan 2008; Wu et al. 2008).
Both cause effects on cell division by damaging the spindle apparatus during mitosis (Burke et al. 2006; Castoldi et al. 2000; Gribble et al. 2005; Kim et al. 2007; Ou et al. 1999b; Machaty et al. 1999; Rodier et al. 1984).
Both MeHg and EtHg bind to the amino acid cysteine (Clarkson 1995; Wu et al. 2008).
Both MeHg and EtHg strongly inhibit the reacylation of arachidonic acid, thus inhibiting the reincorporation of this fatty acid into membrane phospholipids (Shanker et al. 2002; Verity et al. 1994; Zarini et al. 2006).
Both cause an increase in NOS, causing an overproduction of NO (Chen et al. 2003; Chuu et al. 2001; Shinyashiki et al. 1998).
Both disrupt glutamate homeostasis (Farina et al. 2003a, b; Manfroi et al. 2004; Mutkus et al. 2005; Yin et al. 2007).
Both alter intracellular calcium homeostasis (Elferink 1999; Hare et al. 1993;Kang et al. 2006; Limke et al. 2004b; Machaty et al. 1999; Marty and Atchison1997; Minnema et al. 1987; Peng et al. 2002; Sayers et al. 1993; Sirois and Atchison, 2000; Szalai et al. 1999; Tornquist et al. 1999; Zarini et al. 2006).
Both cause effects on receptor binding/neurotransmitter release involving one or more transmitters (Basu et al. 2008; Coccini et al. 2000; Cooper et al. 2003; Fonfria et al. 2001; Ida-Eto et al. 2011; Ndountse and Chan 2008; Yuan and Atchison 2003).
“This study is a nuclear bomb detonating over the CDC,” Boyd Haley, chairman emeritus of the University of Kentucky Chemistry Department, said. “It should be getting international, front page headlines.”

Reviews of Environmental Contamination and Toxicology
Reviews of Environmental Contamination and Toxicology publishes reviews pertaining to the sources, transport, fate and effects of contaminants in the environment. The series provides a place for the publication of critical reviews of the current knowledge and understanding of environmental sciences in order to provide insight into contaminant pathways, fate and behavior in environmental compartments and the possible consequences of their presence, with multidisciplinary contributions from the fields of analytical chemistry, biochemistry, biology, ecology, molecular and cellular biology (in an environmental context), and human, wildlife and environmental toxicology. This book series does not typically consider submissions dealing with technical aspects of occupational exposure and effects in humans, wastewater treatment and effluent characterization, or remediation of contaminated sites. However, submissions addressing one of these topic areas may be considered where there exists a strong link to the receiving environment, and/or the identification of emerging contaminants of concern. All manuscripts will be peer-reviewed by experts in the field. Reviewers will be asked to consider coverage and critical appraisal of the subject, originality , relevance, and impact to the wider scientific community. Authors writing in a second language are encouraged to have their manuscript corrected by a native English speaker or by a professional editing firm. Abstracts, short communications and notes will not be accepted. Where appropriate, such submissions may be referred to our companion journal, the Bulletin of Environmental Contamination and Toxicology (BECT), while full-length research articles are typically the purview of Archives of Environmental Contaminant and Toxicology (AECT). RECT prefers extended reviews of a length including references of more than 5,000 words, and without an upper word count limit. Reviews of shorter length (i.e. where length including references of less than 5,000 words) which may be suitable for case studies, a focused topic or an applied subject of debate or interest can be submitted to AECT. Authors may directly contact the Editor-in-Chief if they wish to clarify which publication is most suited for their submission.

Mainstream News Reporting That #BigPharma Is Paying Everyone Off!
Who would have guessed?

New Study Confirms That Mercury Is Linked to Autism
Robert F. Kennedy, Jr.
Two new studies by international teams, including Egyptian scientists, have validated the link between autism and mercury.
In an article published in the journal Metabolic Brain Disease, a team of nine scientists from leading Egyptian universities and medical schools confirmed the causal role of mercury in the onset of autism.
The scientists determined the extent of mercury poisoning in children by measuring urinary excretion of organic compounds called porphyrins, which act as biomarkers for mercury toxicity. The researchers also measured blood levels of mercury and lead. The researchers found a strong relationship between mercury toxicity and the presence of autism and a direct correlation between levels of mercury toxicity and the severity of autism symptoms.
The scientists studied 100 children; 40 with autism spectrum disorder (ASD), 40 healthy individuals and 20 healthy siblings of ASD children. The results showed that the children with ASD had significantly higher mercury levels than healthy children and healthy siblings. Children with the highest mercury levels had the most severe autism symptoms.
At least six American studies have linked autism presence or severity to mercury exposure as determined by measuring urinary porphyrins. The first study, completed by Heyer et al. in 2012 (Autism Res 5:84) showed a correlation between the presence of autism and specific urinary porphyrins associated with mercury toxicity. This affirmed an earlier study by Kern et al. (2011, Pediatr Int 53:147) where specific porphyrins associated with mercury toxicity were significantly higher in ASD children as compared to non-autistic controls. Woods et al. (2010, Environ Health Perspect 118:1450) also saw disordered porphyrin metabolism in autistic kids which was not observed in non-autistic control children. This again suggested increased mercury toxicity associated with autism and autism spectrum disorder.

Study – Altered urinary porphyrins and mercury exposure as biomarkers for autism severity in Egyptian children with autism spectrum disorder
Abstract
Autism spectrum disorder (ASD) is a complex neurodevelopmental disorder that affects social, communication, and behavioral development. Recent evidence supported but also questioned the hypothetical role of compounds containing mercury (Hg) as contributors to the development of ASD. Specific alterations in the urinary excretion of porphyrin-containing ring catabolites have been associated with exposure to Hg in ASD patients. In the present study, the level of urinary porphyrins, as biomarkers of Hg toxicity in children with ASD, was evaluated, and its correlation with severity of the autistic behavior further explored. A total of 100 children was enrolled in the present study. They were classified into three groups: children with ASD (40), healthy controls (40), and healthy siblings of the ASD children (20). Children with ASD were diagnosed using DSM-IV-TR, ADI-R, and CARS tests. Urinary porphyrins were evaluated within the three groups using high-performance liquid chromatography (HPLC), after plasma evaluation of mercury (Hg) and lead (Pb) in the same groups. Results showed that children with ASD had significantly higher levels of Hg, Pb, and the porphyrins pentacarboxyporphyrin, coproporphyrin, precoproporphyrin, uroporphyrins, and hexacarboxyporphyrin compared to healthy controls and healthy siblings of the ASD children. However, there was no significant statistical difference in the level of heptacarboxyporphyrin among the three groups, while a significant positive correlation between the levels of coproporphyrin and precoproporphyrin and autism severity was observed. Mothers of ASD children showed a higher percentage of dental amalgam restorations compared to the mothers of healthy controls suggesting that high Hg levels in children with ASD may relate to the increased exposure to Hg from maternal dental amalgam during pregnancy and lactation. The results showed that the ASD children in the present study had increased blood Hg and Pb levels compared with healthy control children indicating that disordered porphyrin metabolism might interfere with the pathology associated with the autistic neurologic phenotype. The present study indicates that coproporphyrin and precoproporhyrin may be utilized as possible biomarkers for heavy metal exposure and autism severity in children with ASD.

CDC Knew Its Vaccine Program Was Exposing Children to Dangerous Mercury Levels Since 1999
Robert F. Kennedy, Jr. and Lyn Redwood, RN, MSN
Jan. 18, 2017 08:12PM EST
Uncovered documents show that the U.S. Food and Drug Administration (FDA) and the Centers for Disease Control and Prevention (CDC) knew that infant vaccines were exposing American children to mercury far in excess of all federal safety guidelines since 1999. The documents, created by a FDA consulting toxicologist, show how federal regulators concealed the dangerous impacts and lied to the public.
PDF source
In 1997, Congress passed the FDA Modernization Act. A provision of that statute required the FDA to “compile a list of drugs that contain intentionally introduced mercury compounds, and provide a quantitative and qualitative analysis of the mercury compounds on the list.” In response, manufacturers reported the use of the mercury-based preservative, thimerosal, in more than 30 licensed vaccines.
FDA’s Center for Biologics Evaluation and Research (CBER) was responsible for adding up the cumulative exposure to mercury from infant vaccines, a simple calculation that, astonishingly, had never been performed by either the FDA or the CDC. When the agency finally performed that basic calculation, the regulators realized that a six month-old infant who received thimerosal-preserved vaccines following the recommended CDC vaccine schedule would have received a jaw dropping 187.5 micrograms of mercury.
Instead of immediately ordering the removal of thimerosal, FDA officials circled the wagons treating the public health emergency as a public relations problem. Peter Patriarca, then director of the FDA Division of Viral Products, warned his fellow bureaucrats that hasty removal of thimerosal from vaccines would:
” … raise questions about FDA being ‘asleep at the switch’ for decades by allowing a potentially hazardous compound to remain in many childhood vaccines, and not forcing manufacturers to exclude it from new products. It will also raise questions about various advisory bodies regarding aggressive recommendations for use. We must keep in mind that the dose of ethylmercury was not generated by “rocket science.” Conversion of the percentage thimerosal to actual micrograms of mercury involves ninth grade algebra. What took the FDA so long to do the calculations? Why didn’t CDC and the advisory bodies do these calculations when they rapidly expanded the childhood immunization schedule?”
The agency consulted with experts in the field of toxicology to better understand the potential impact of these exposure levels. One consultant was Barry Rumack, MD. Dr. Rumack, at the time, had a private consulting practice, Rumack Consulting, where he offered “toxicologic and pharmacologic evaluation of drugs, biological and potentially toxic or hazardous agents for government and industry.” After creating several scenarios based on infants’ ages and weights, Dr. Rumack modeled blood and body burden levels in 1999.

Local authority is granted permission by a top judge to forcibly vaccinate a seven-month-old baby boy against his mother’s wishes despite claims she has bad reactions to jabs in the past
The London Borough of Barnet can vaccinate the seven-month-old baby boy
The boy’s mother says her other children had bad reactions from immunisations
But Barnet said potential consequence of not immunising was too great to risk
Mr Justice MacDonald has said that vaccination is in the boy’s best interests
The jabs will protect the baby against bacterial infections which can lead to highly dangerous forms of meningitis

November 7, 2002
Vaccination Safety, Part 1 In the first part of a day-long conference on the safety of various vaccination programs, participants talked about possible adverse effects of certain vaccines, the scientific community’s response to potential problems, public education efforts and the viability of mass, mandatory vaccination programs

Missouri Bill Would Ban Mercury Vaccines; First Step to Nullify Federal Policy in Practice
JEFFERSON CITY, Mo. (Jan. 31, 2017) – A Missouri House bill would ban public health clinics from administering vaccines that contain mercury or any other metal put into the vaccine for preservation purposes, contradicting approved federal policy.
House Bill 331 (HB331) was introduced by Rep. Lynn Morris (R-Nixa) to mitigate concerns regarding vaccine safety. With the exception of health emergencies determined by the Department of Health & Senior Services with concurrence from the governor, the following provision would apply:
Beginning August 28, 2018, no vaccine containing mercury or other metal for preservation or other purpose shall be administrated to a child or adult in a public health clinic in Missouri.
HB331 begins the process of nullifying potential vaccine mandates, which generally have their basis in federal recommendations or guidelines from the Centers for Disease Control and Prevention (CDC). Although these federal rules are not technically binding, they often influence policy-makers and individuals at the local and state levels to adopt coercive mandates regarding mercury-laced vaccines and other toxic substances.
By taking the rule-making power back into their own hands, the state of Missouri can disconnect from federal control and restore its sovereignty on this key issue.
NECESSITY
Measures such as HB331 push back against federal narratives regarding immunizations. The Food and Drug Administration (FDA) downplays concerns regarding the use of thimerosal, a preservative containing mercury. They even admit on their own website that mercury is still being used to preserve certain vaccines:
Thimerosal, which is approximately 50% mercury by weight, has been one of the most widely used preservatives in vaccines… While the use of mercury-containing preservatives has declined in recent years with the development of new products formulated with alternative or no preservatives, thimerosal has been used in some immune globulin preparations, anti-venins, skin test antigens, and ophthalmic and nasal products, in addition to certain vaccines.
As the FDA downplays the concerns related to thimerosal and mercury in vaccines, whistle-blowers are singing a different tune. The National Vaccine Information Center, a non-profit watchdog organization, reports that the threat is still alarming – especially pertaining to infants:
Most infants are still routinely given Thimerosal-containing influenza vaccine even though there are Thimerosal-free and vaccines with trace amounts of Thimerosal. Infants receiving a Thimerosal-containing influenza vaccine are dosed at 6 months with 12.5 mcg of ethyl mercury and at 7 months with an additional 12.5 mcg. Adult Thimerosal-containing vaccines contain roughly 25mcg.
The CDC aids the FDA in promulgating their point of view. Although the CDC attempts to maintain a veneer of independence and credibility, there are facts showing that narrative to be false. The CDC Foundation boasts that it helps the CDC “do more, faster.” It is able to do this because the CDC Foundation receives annual funding from a host of corporations including Pharmaceutical giants Merck, Roche, and Emergent BioSolutions Inc.

Vaccines, Retroviruses, DNA, and the Discovery That Destroyed Judy Mikovits’ Career

Vaccines, Retroviruses, DNA, and the Discovery That Destroyed Judy Mikovits’ Career
In 2011, she made the discovery that destroyed her career. She found that at least 30% of our vaccines are contaminated with gammaretroviruses. Not only is this contamination associated with autism and chronic fatigue syndrome, it is also associated with Parkinson’s, Lou Gehrig’s disease, and Alzheimer’s.
When she released this shocking information, she was warned by Dr. Andrew Wakefield that she would become a target, just as he had been. But she assured him that all of her work had been properly reviewed and, of course, she was safe.
She was wrong. She was threatened and told to destroy her data; she refused. She was fired, then arrested for supposedly stealing her data from her worksite. She had been facing charges and was bound by a gag order from the court for the last four years. Recently, charges were dropped and the gag order was lifted. Dr. Mikovits is now free to talk, and boy is she talking.
The retroviruses contaminating vaccines originate from mice used for research. Dr. Mikovits asks, “How many new retroviruses have we created through all the mouse research, the vaccine research, gene therapy research? More importantly, how many new diseases have we created?”
“When they destroyed all of our work, and discredited everything I or Frank Ruscetti had ever published, and arranged for the publication of my mug shot in Science, the NIH very deliberately sent the message to researchers everywhere about what would happen to any honest scientist who dared ask those important questions.”
New technology now exists to clean up retroviruses in vaccines and blood. Dr. Mikovits believes we will win this war, that we will eventually clean up vaccines, stop vaccinating infants, and stop injecting our children with multiple vaccines. But she also believes the government will continue to cover up their culpability in the current epidemic of autism and other diseases.
When asked about vaccine-injured children, she says, “Those are the victims. And that’s why, I’m working and why, you know, I’ll never shut up. Because they’re the victims that have been hung out to dry.”

300 pages of study abstracts culled directly from the National Library of Medicine’s pubmed.gov

GreenMedInfo.com has painstakingly collected over 300 pages of study abstracts culled directly from the National Library of Medicine’s pubmed.gov bibliographic database on the wide-ranging adverse health effects linked to vaccines in the today’s schedule (over 200 distinct adverse effects, including death), as well as numerous studies related to vaccine contamination, and vaccine failure in highly vaccine compliant populations.
1. Hepatitis B Vaccination of Male Neonates and Autism Annals of Epidemiology, September 2009 CM Gallagher, MS Goodman, Stony Brook University Medical CenterBoys vaccinated as neonates had threefold greater odds for autism diagnosis compared to boys never vaccinated or vaccinated after the first month of life.
2. Porphyrinuria in childhood autistic disorder: Implications for environmental toxicity Toxicology and Applied Pharmacology, 2006 Robert Natafa, et al, Laboratoire Philippe Auguste, Paris, France These data implicate environmental toxicity in childhood autistic disorder.
3. Theoretical aspects of autism: Causes—A review Journal of Immunotoxicology, January-March 2011 Helen V. Ratajczak, PhD Autism could result from more than one cause, with different manifestations in different individuals that share common symptoms. Documented causes of autism include genetic mutations and/or deletions, viral infections, and encephalitis following vaccination.
4. Uncoupling of ATP-mediated Calcium Signaling and Dysregulated IL-6 Secretion in Dendritic Cells by Nanomolar Thimerosal Environmental Health Perspectives, July 2006. Samuel R. Goth, Ruth A. Chu Jeffrey P. Gregg This study demonstrates that very low-levels of Thimerosal can contribute to immune system disregulation.
5. Gender-selective toxicity of thimerosal Exp Toxicol Pathol. 2009 Mar;61(2):133-6. Epub 2008 Sep 3. Branch DR, Departments of Medicine and Laboratory Medicine and Pathobiology, University of Toronto A recent report shows a correlation of the historical use of thimerosal in therapeutic immunizations with the subsequent development of autism; however, this association remains controversial. Autism occurs approximately four times more frequently in males compared to females; thus, studies of thimerosal toxicity should take into consideration gender-selective effects. The present study was originally undertaken to determine the maximum tolerated dose (MTD) of thimersosal in male and female CD1 mice. However, during the limited MTD studies, it became apparent that thimerosal has a differential MTD that depends on whether the mouse is male or female.
6. Comparison of Blood and Brain Mercury Levels in Infant monkeys exposed to Vaccines Containing Thimerosal Environmental Health Perspectives, Aug 2005. Thomas Burbacher, PhD, University of Washington This study demonstrates clearly and unequivocally that ethyl mercury, the kind of mercury found in vaccines, not only ends up in the brain, but leaves double the amount of inorganic mercury as methyl mercury, the kind of mercury found in fish. This work is groundbreaking because little is known about ethyl mercury, and many health authorities have asserted that the mercury found in vaccines is the “safe kind.” This study also delivers a strong rebuke of the Institute of Medicine’s recommendation in 2004 to no longer pursue the mercury-autism connection.
7. Increases in the number of reactive glia in the visual cortex of Macaca fascicularis following subclinical long-term methyl mercury exposure Toxicology and Applied Pharmacology, 1994 Charleston JS et al, Department of Pathology, School of Medicine, University of Washington The identities of the reactive glial cells and the implications for the long-term function and survivability of the neurons due to changes in the glial population following subclinical long-term exposure to mercury are discussed.
8. Neuroglial Activation and Neuroinflammation in the Brain of Patients with Autism Annals of Neurology, Feb 2005. Diana L. Vargas, MD [Johns Hopkins University] This study, performed independently and using a different methodology than Dr. Herbert (see above) reached the same conclusion: the brains of autistic children are suffering from inflammation.
9. Autism: A Brain Disorder, or a Disorder That Affects the Brain? Clinical Neuropsychiatry, 2005 Martha R. Herbert M.D., Ph.D., Harvard University Autism is defined behaviorally, as a syndrome of abnormalities involving language, social reciprocity and hyperfocus or reduced behavioral flexibility. It is clearly heterogeneous, and it can be accompanied by unusual talents as well as by impairments, but its underlying biological and genetic basis in unknown. Autism has been modeled as a brain-based, strongly genetic disorder, but emerging findings and hypotheses support a broader model of the condition as a genetically influenced and systemic.
10. Activation of Methionine Synthase by Insulin-like Growth Factor-1 and Dopamine: a Target for Neurodevelopmental Toxins and Thimerosal  Molecular Psychiatry, July 2004. Richard C. Deth, PhD [Northeastern University] This study demonstrates how Thimerosal inhibits methylation, a central driver of cellular communication and development.
11. Validation of the Phenomenon of Autistic Regression Using Home Videotapes Archives of General Psychiatry, 2005 Emily Werner, PhD; Geraldine Dawson, PhD, University of WashingtonConclusion This study validates the existence of early autistic regression.
12. Blood Levels of Mercury Are Related to Diagnosis of Autism: A Reanalysis of an Important Data Set Journal of Child Neurology, 2007 M. Catherine DeSoto, PhD, Robert T. Hitlan, PhD -Department of Psychology, University of Northern Iowa Excerpt: “We have reanalyzed the data set originally reported by Ip et al. in 2004 and have found that the original p value was in error and that a significant relation does exist between the blood levels of mercury and diagnosis of an autism spectrum disorder. Moreover, the hair sample analysis results offer some support for the idea that persons with autism may be less efficient and more variable at eliminating mercury from the blood.”
13. Developmental Regression and Mitochondrial Dysfunction in a Child With Autism Journal of Child Neurology, February 2006 Jon S. Poling, MD, PhD, Department of Neurology and Neurosurgery, Johns Hopkins Hospital Excerpt: “Children who have (mitochondrial-related) dysfunctional cellular energy metabolism might be more prone to undergo autistic regression between 18 and 30 months of age if they also have infections or immunizations at the same time.”
14. Oxidative Stress in Autism: Elevated Cerebellar 3-nitrotyrosine Levels American Journal of Biochemistry and Biotechnology, 2008 Elizabeth M. Sajdel-Sulkowska, – Dept of Psychiatry, Harvard Medical School Excerpt: The preliminary data suggest a need for more extensive studies of oxidative stress, its relationship to the environmental factors and its possible attenuation by antioxidants in autism.”
15. Large Brains in Autism: The Challenge of Pervasive Abnormality The Neuroscientist, 2005. Martha Herbert, MD, PhD, Harvard University This study helps refute the notion that the brains of autistic children are simply wired differently and notes, “neuroinflammation appears to be present in autistic brain tissue from childhood through adulthood.” Dr. Herbert suggests that chronic disease or an external environmental source (like heavy metals) may be causing the inflammation.
16. Evidence of Toxicity, Oxidative Stress, and Neuronal Insult in Autism Journal of Toxicology and Environmental Health, Nov-Dec 2006. Janet Kern, Anne Jones, Department of Psychiatry, University of Texas Southwestern Medical Center “This article discusses the evidence for the case that some children with autism may become autistic from neuronal cell death or brain damage sometime after birth as result of insult; and addresses the hypotheses that toxicity and oxidative stress may be a cause of neuronal insult in autism… the article discusses what may be happening over the course of development and the multiple factors that may interplay and make these children more vulnerable to toxicity, oxidative stress, and neuronal insult.”
17. Oxidative Stress in Autism Pathophysiology, 2006. Abha Chauhan, Ved Chauhan This study provides a helpful overview of the growing evidence supporting the link between oxidative stress and autism.
18. Thimerosal Neurotoxicity is Associated with Glutathione Depletion: Protection with Glutathione Precursors Neurotoxicology, Jan 2005. S. Jill James, PhD, University of Arkansas This recent study demonstrates that Thimerosal lowers or inhibits the body’s ability to produce Glutathione, an antioxidant and the body’s primary cellular-level defense against mercury.
19. Aluminum adjuvant linked to gulf war illness induces motor neuron death in mice Neuromolecular Medicine, 2007 Christopher Shaw, Ph.D., Department of Ophthalmology and Program in Neuroscience, University of British Columbia This study demonstrates the extreme toxicity of the aluminum adjuvant used as a preservative in vaccines.
20. Environmental mercury release, special education rates, and autism disorder: an ecological study of Texas Health & Place, 2006 Raymond F. Palmer, University of Texas Health Science Center This study demonstrated the correlation between environmental mercury and autism rates in Texas.
21. Autism Spectrum Disorders in Relation to Distribution of Hazardous Air Pollutants in the SF Bay Area Environmental Health Perspectives, September, 2006 Gayle Windham, Div. of Environmental and Occupational Disease Control, California Department of Health Services Excerpt: “Our results suggest a potential association between autism and estimated metal concentrations, and possibly solvents, in ambient air around the birth residence.”
22. A Case Series of Children with Apparent Mercury Toxic Encephalopathies Manifesting with Clinical Symptoms of Regressive Autistic Disorder Journal of Toxicology and Environmental Health, 2007 David A. Geier, Mark R. Geier This study reviewed the case histories and medical profiles of nine autistic children and concluded that eight of the nine children were mercury toxic and this toxicity manifested itself in a manner consistent with Autism Spectrum Disorders.
23. Attention-deficit hyperactivity disorder and blood mercury level: a case-control study in chinese children Neuropediatrics, August 2006 – P.R. Kong Excerpt: “There was significant difference in blood mercury levels between cases and controls, which persists after adjustment for age, gender and parental occupational status. The geometric mean blood mercury level was also significantly higher in children with inattentive and combined subtypes of ADHD. High blood mercury level was associated with ADHD. Whether the relationship is causal requires further studies.”
24. The Changing Prevalence of Autism In California Journal of Autism and Developmental Disorders, April 2003 Mark F. Blaxill, David S. Baskin, and Walter O. Spitzer This study helps to refute the supposition made by some researchers that autism’s epidemic may only be due to “diagnostic substitution”.
25. Mitochondrial Energy-Deficient Endophenotype in Autism American Journal of Biochemistry and Biotechnology 2008 J. Jay Gargus and Faiqa Imtiaz, School of Medicine, University of California, Irvine, “While evidence points to a multigenic etiology of most autism, the pathophysiology of the disorder has yet to be defined and the underlying genes and biochemical pathways they subserve remain unknown.”
26. Bridging from Cells to Cognition in Autism Pathophysiology: Biological Pathways to Defective Brain Function and Plasticity American Journal of Biochemistry and Biotechnology 2008 Matthew P. Anderson, Brian S. Hooker and Martha R. Herbert, Cambridge Health Alliance/Harvard Medical School/Beth Israel Deaconess Medical Center “We review evidence to support a model where the disease process underlying autism may begin when an in utero or early postnatal environmental, infectious, seizure, or autoimmune insult triggers an immune response that increases reactive oxygen species (ROS) production in the brain that leads to DNA damage (nuclear and mitochondrial) and metabolic enzyme blockade and that these inflammatory and oxidative stressors persist beyond early development (with potential further exacerbations), producing ongoing functional consequences.”
27. Heavy-Metal Toxicity—With Emphasis on Mercury John Neustadt, ND, and Steve Pieczenik, MD, PhD Conclusion: Metals are ubiquitous in our environment, and exposure to them is inevitable. However, not all people accumulate toxic levels of metals or exhibit symptoms of metal toxicity, suggesting that genetics play a role in their potential to damage health.
28. Evidence of Mitochondrial Dysfunction in Autism and Implications for Treatment American Journal of Biochemistry and Biotechnology Daniel A. Rossignol, J. Jeffrey Bradstreet MtD and oxidative stress may also explain the high male to female ratio found in autism due to increased male vulnerability to these dysfunctions.
29. Proximity to point sources of environmental mercury release as a predictor of autism prevalence
Health & Place, 2008 Raymond F. Palmer et al, University of Texas Health Science Center This study should be viewed as hypothesis-generating – a first step in examining the potential role of environmental mercury and childhood developmental disorders. Nothing is known about specific exposure routes, dosage, timing, and individual susceptibility. We suspect that persistent low-dose exposures to various environmental toxicants, including mercury, that occur during critical windows of neural development among genetically susceptible children (with a diminished capacity for metabolizing accumulated toxicants) may increase the risk for developmental disorders such as autism.
30. Epidemiology of autism spectrum disorder in Portugal: prevalence, clinical characterization, and medical conditions Developmental Medicine & Child Neurology, 2007 Guiomar Oliveira MD PhD et al, Centro de Desenvolvimento da Criança, Hospital Pediátrico de Coimbra; Assunção Ataíde BSc, Direcção Regional de Educação do Centro Coimbra; The objective of this study was to estimate the prevalence of autistic spectrum disorder (ASD) and identify its clinical characterization, and medical conditions in a paediatric population in Portugal.